Download PDF by George F. Vande Woude, George Klein: Advances in Cancer Research, Vol. 77

By George F. Vande Woude, George Klein

ISBN-10: 0120066777

ISBN-13: 9780120066773

Advances in melanoma examine offers important info at the intriguing and fast-moving box of melanoma learn. right here once more, impressive and unique studies are provided. Key positive aspects * The Yin Yang of TCF/b-Catenin Signalling * Biochemical and scientific Implications of the Erb/HER Signaling community of progress issue Receptors * p53 and Human melanoma: the 1st 10000 Mutations * Macrophage Stimulating Protein * CD44 Glycoproteins in Colorectal melanoma; Expression; functionality; and Prognostic price * an easy Modes for Carcinogenesis of Colorectal melanoma with Microsatellite Instability

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Additional resources for Advances in Cancer Research, Vol. 77

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Bertheau et al. (1998) showed that the status of ErbB-2 was prognostic by the Cox model analysis in the younger patient cohort, under age 35, whereas p53 was prognostic in the age 36–50 group. , 1991). , 1998). , 1991). , 1990). These and other conflicting results may relate to the size of the population analyzed and to some technical considerations. Essentially, determination of positivity may vary, especially in earlier studies, with the type of analysis or the identity of antibody used for ErbB2 immunostaining.

Development (Cambridge, UK) 120, 369–380. Polakis, P. (1997). Biochim. Biophys. Acta 1332, 127–147. , and Polakis, P. (1997). Oncogene 15, 2833–2839. , and Bienz, M. (1997). ) 88, 777–787. , and Mello, C. (1997). ) 90, 707–716. , and Clevers, H. (1998). Nature (London) 395, 608–612. , Chamberlain, S. , Masiarz, F. , and Polakis, P. (1993). Science 262, 1731–1734. , and Polakis, P. (1996). Science 272, 1023–1026. , and Polakis, P. (1997). Science 275, 17990–1792. , and Melton, D. (1991). ) 67, 741–752.

HOW DOES ErbB-2 INDUCE CANCER? A. In Vitro Transforming Potential of ErbB-2 How ErbB-2 causes cancer is a question that has been repeatedly asked— and the answers reevaluated—during the past 15 years. Its role in cellular transformation, insinuated by its abundance in a wide range of human tumors, is supported by a few but solid lines of evidence that therapies directed against this receptor can indeed impede tumor growth. , 1991). , 1987), indicating that a threshold level of overexpression is crucial for its oncogenic potential.

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Advances in Cancer Research, Vol. 77 by George F. Vande Woude, George Klein


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